[dropcap]Y[/dropcap]our next patient is a 32-year-old woman complaining of a headache. You check her vitals: a heart rate of 101 bpm, blood pressure of 150/90 mm Hg, sat of 100%, respirations of 22 bpm, and a 10/10 on the pain scale. You talk to her about her complaint of headache. She says she is allergic to Tylenol.
The patient is calm, healthy-looking, and in no distress. You do your usual head-to-toe review, and ask your typical history and physical questions. Her neurological exam is negative. She is positive for a wide range of complaints—body aches, crampy back pain, runny nose, slight abdominal pain, maybe some diarrhea, and of course, the headache. You spend minimal time on the other systems, and inquire about her main complaint. Because this patient has a plethora of issues and is of child-bearing age, you check her CBC, BMP, ECG, flu, UA, and hCG. You know they are going to come back negative. You consider a D-dimer because she is on birth control and might be PERC-positive, but you’re not convinced this is PE-related.
What if we start the conversation differently? What if we did an HPI with specific questions? She has been to the ED before, and doesn’t have a primary care physician. Nothing stands out as an emergency. You wonder if a workup is necessary, but don’t want to miss any zebras. A few clues suggest this could be something nefarious, but the opioid epidemic should tell us otherwise.
The bottom line is patients want pain medications. We have to figure out if a patient is drug-seeking or has a valid complaint. The problem is we cannot predict what she has based on a limited history and physical. The answer to this case lies in our bedside manner, approach, and honesty. Opioid abuse, misuse, withdrawal, and addiction are not diagnoses of exclusion; none of us wants all those tests done.
ED visits and overdose deaths involving opioid analgesics and benzodiazepines increased from 0.6 to 1.7 per 100,000 between 2004 and 2011. (Am J Prev Med 2015;49:493.) The number of deaths per year attributed to prescription opioid medications reached 16,651 in 2010. (MMWR 2013;62:234; http://bit.ly/2yM9QfS.)
We don’t ignore the one or two percent risk of certain diagnoses like PE, MI, and aortic dissection, so why ignore the signs of drug-seeking? The abuse of and addiction to opioids is a serious global problem that affects the health, social, and economic welfare of all societies. (National Institutes of Health, 2014; http://bit.ly/2yPhFUg.) Just like we would not forget to ask about smoking or cholesterol levels in a patient with chest pain, we cannot forget to ask about pain medication use.
It is estimated that between 26.4 and 36 million people abuse opioids worldwide. (United Nations Office on Drugs and Crime, 2012; http://bit.ly/2yQcYJU.) An estimated 2.1 million Americans suffered from substance abuse disorders related to prescription opioid pain relievers in 2012, and 467,000 people are addicted to heroin. (Substance Abuse and Mental Health Services Administration, 2013; http://bit.ly/2yRKaAw.) Surely, the smaller percentage of those with PE, MI, or dissection is outweighed by a diagnosis of opioid abuse or withdrawal.
A study in the Western Journal of Emergency Medicine looked at 178 patients from the case management program who made 2,486 visits in one year. (2012;13:416.) Headache accounted for 21.7 percent and back pain 20.8 percent, while requesting a refill took up seven percent, lost or stolen medication 0.6 percent, 10/10 pain 29.1 percent, greater than 10/10 pain 1.8 percent, and out of medication 9.5 percent. Not every headache is going to be a stroke.
Just as we take measures to rule out PE, dissection, and MI by risk-stratifying patients, we should consider an individual’s risk for abuse before prescribing opioids. (Ann Emerg Med 2016;68:S81.) We are behind the times if we do not risk-stratify patients for potential abuse or misuse. Narcotics-seeking is often hidden among the initial investigation. It may even get lost in a triage provider’s initial questions and three to four hours of unnecessary tests. Neither the provider nor the patient really wants to talk about narcotic abuse, but we should be asking our patients flat-out: Do you have a problem with narcotic pain medication use?
Addiction has a strong, visceral hold on our patients, and we are ignoring the easy stuff. Questioning our patients directly about it should not be a game. When all the information is gathered in the initial interview without bias or judgment, patients are more likely to get the treatment they need. This may stop us all from doing unnecessary workups and costly imaging.
Next time we interview a patient, we should ask if he has tried anything for pain and list multiple medications: acetaminophen, ibuprofen, tramadol, morphine, or methadone. Patients may not be upfront about these medications if they have true addiction issues. They are fearful we will not prescribe pain medications or treat their acute pain. When we set the standard of care in the beginning, however, we can avoid drama and overtesting.
We should ask every patient of every age and walk of life about his personal experience with pain and then consider how to address it. This starts with being upfront in a nonjudgmental way and using a team approach to their care. It may prevent thousands of deaths and save hospitals millions of dollars. It will also get patients the help they actually need. The nature of their complaint doesn’t even matter, but our practices can change because of it.
Share this article on Twitter and Facebook.
Access the links in EMN by reading this on our website or in our free iPad app, both available at www.EM-News.com.
Name: Jeffrey Randa
Age at time of Dissection: 48
Type of Dissection: Both Ascending and Descending
Date of Aortic Dissection: 8 December 2011
Tell Us Your Story:
[dropcap]T[/dropcap]his is a follow up to my first post about 6 years ago. Re-reading it, I am humbled by how humbled I was as I wrote it, and reminded of how lucky it is to survive this trauma. I am glad to report that my life has almost entirely returned to normal over the last 6 years, despite the severity of my dissection.
Bit by bit, over the years, I have returned to weightlifting, although I have learned to do it much differently than before (no “Valsalva” maneuver), taking the time to breathe through every rep. My cardio conditioning is fair to good, meaning better than average for my age, but probably not as good as it should be given my AD; I’m working on it.
And that’s really the point of my post. It seems that many people find this site soon after they have surgery. I was wounded when I made my first submission, and even my typing suffered as I wrote it. I thought I’d never be strong again and that I was going to forever be hobbled.
When I finally did make it back to my weight bench, the 45 pound bar felt like a ton. I’m not out to get huge or lift tons of weight anymore, and even now, as I age, I’ve backed off a bit, but a few years ago I was able to dead lift 350 lbs and breathe through it rather easily.
At one point, my cardio conditioning was much better (that tends to go in cycles) and I could run a few miles without killing myself. I can still outrun and outlast almost anyone my age.
As I recovered from my surgery, I thought I would be frail and weak forever. That’s not necessarily the case, and you MUST make the best of what you have. Never give up, quit or otherwise surrender. Fight to be the fittest and strongest you
can. Nothing good will come from just sitting, or, worse yet, just sitting and eating all the wrong foods.
I do think it’s important to take one’s time, but also to work at becoming fit. Walk. Walk a lot. Do some strength training. Eat well and enjoy life, but do it intelligently (everything in moderation (including moderation) remember?).
As I sit here now, I cannot think of any limitations that my AD has imposed on my life or lifestyle. I think one should live fully, albeit mindfully, at that.
In the early days of my recovery, I was taking every day as a gift. I sometimes lose sight of that now, at least for a bit, but then I remind myself how lucky I am to be here, how easy it is to be gone, and the wisdom of the old adage, “Don’t sweat the small stuff because it’s all small stuff.”
Carry on, my friends. Remember, the best revenge is to live well…
[dropcap]H[/dropcap]ello – I had a Type A aortic dissection repair August 31, 2017 at Baylor in Dallas. My surgeon, Juan MacHannaford, told me the dissection was from top to bottom. I was flown there from Pampa, TX due to the wonderful diagnostic abilities of my cardiologist in our local emergency room.
His quick action saved my life. I had a history of untreated high blood pressure for approximately 3-5 years before beginning treatment in October, 2013. When he saw me in the ER, I had experiences sever stabbing pain from my throat down to my stomach and almost immediately lost feeling in my legs. I also eventually had vomiting and lost consciousness.
My surgeon almost immediately from visual examination and I am not sure what other indicators, told me when I saw him a week after being released from the hospital, he felt 99% sure I have Marfans Syndrome. I am female, 6′ tall with a long torso, arms and legs.
I had a great uncle who had a aortic aneurysm in the 1960’s. That is the only family history I am aware of. Both of my parents are deceased and there are no other living relatives to talk to about this. I have had retina detachments in both eyes and a vitriol hemorrhage in one eye, all happening within 8 to 10 years intervals, starting when I was in my mid 30’s. I have two sons who are 6’2″ and 6’5″.
The surgeon has recommended genetic testing for them. I have changed my eating habits more than what they were, increased my exercise and made other life style changes, all thanks to a wonderful cardiac rehab program I have been attending since the surgery.
I will have a CT scan in 6 months and then once a year for the rest of my life. My scan October 26th, showed 3.2 in my abdomen and 3.4 in my chest. I know this is not something I should think about a lot, but I feel good about my future as long as I keep my scans up.
Are aneurysms common for someone in my situation? Thank you for any input you can give.
Answer from Dr. Liang:
I would certainly agree with your surgeons concerns about Marfan syndrome as a cause cause of the dissection. After the dissection the part of the aorta that is dissected beyond the repair is at increased risk for aneurysm formation so the regular CT’s recommended by your surgeon make sense. We also recommend very rigorous blood pressure control at this point to reduce the stress on the aorta.
Getting evaluated for a genetic cause of the dissection such as Marfan syndrome is very important, especially since you have children. If you can I would recommend seeing Dianna Milewicz at UT Houston.
TCTMD journalists asked a range of experts about what developments rocked the field this year—or at least made it tremble. Here’s what they had to say.
[dropcap]I[/dropcap]n the last few weeks of 2017, the entire TCTMD news team reached out to a range of cardiologists around the world to ask what they see as the biggest studies, the most auspicious advances, or the most conspicuous flops of the year gone by. Here’s what they had to say.
Coronary Artery Disease
No surprise: several of the physicians we spoke with mentioned the ORBITA trial. “Interesting and provocative,” Allen Jeremias, MD (St. Francis Hospital, Roslyn, NY), called the trial results, which have met with robust debate since they were reported at TCT 2017. Jeremias said he would be interested in seeing an analysis of the roughly three-quarters of patients who had abnormal physiology at baseline and who might derive a benefit from PCI.
ORBITA was on the list for Andrew Foy, MD (Penn State Hershey Medical Center, PA), as well. “I would have been one of those people that would have hypothesized that a lot of the subjective benefit of PCI would not be present in a sham-controlled trial, but it was great that this trial was able to be done. It really shows that interventions aimed at improving subjective endpoints need to be subject to rigorous study. It would be nice if we would do these studies before the widespread adoption of the service as opposed to after the fact.”
Foy said ORBITA should be a game-changer, but doesn’t think it will be, unfortunately. “At least it gets people thinking though, that when it comes to these things, maybe we should be doing more sham-controlled trials and that it’s not unethical,” he said. “In fact, it’s really helpful.”
While ORBITA seemed to steal the show in the last 2 months of the year, Jeremias argued that the DEFINE-FLAIR and iFR-SWEDEHEART trials, showing that instantaneous wave-free ratio (iFR) was noninferior to fractional flow reserve (FFR) for guiding revascularization decisions in intermediate lesions, were arguably the bigger story in CAD this year. Jeremias said he expects to see a lot of interesting subanalyses in the coming year, but believes the trials have already had an impact.
“I’ve been using both for quite some time, but I’ve spoken to many people and they basically completely abandoned FFR and now switched over to iFR, which I think might be a little bit of a strong statement,” he said. “There’s a role for both but in terms of usability, iFR certainly is more user-friendly. It’s quicker, with less side effects because you don’t have to give adenosine. So it’s a more appealing concept for sure.”
The two iFR/FFR trials also got the nod from Ik-Kyung Jang, MD, PhD (Massachusetts General Hospital, Boston, MA), who told TCTMD that at his center, FFR has been almost completely replaced by iFR. “When those two large trials were done independently and showed almost superimposable data, it was very convincing,” Jang said.
Jeremias said important developments also occurred in the areas of complete revascularization for patients with acute MI complicated by cardiogenic shock (CULPRIT-SHOCK), left main PCI, and bioresorbable scaffolds. The latter weathered its share of alarmist headlines in 2017 and was ultimately taken off the market.
“It’s a disappointment,” Jang said, “but I don’t think it’s dead. I think the better second-generation versions will come shortly.”
Joanna Wykrzykowska, MD (Academic Medical Center, Amsterdam, the Netherlands), said the disappointing results of bioresorbable scaffolds shifted focus back to metallic DES. “When everybody realized . . . that the bioresorbable scaffolds were not ready for prime time and were not going to bring down the risk of stent thrombosis to zero as promised, I think we all started rethinking metallic drug-eluting stents.
“The question in everybody’s mind,” she said, “is whether or not there are DES platforms where you can afford to reduce DAPT—to decrease the bleeding risk when somebody has to operate or because the patient has a high risk of bleeding—without the additional risk of stent thrombosis. This is the thinking behind a lot of the ongoing trials.”
Wykrzykowska cited several DAPT de-escalation trials as the “big news” of 2017, including CHANGE DAPT and PRAGUE-18.
“We see in clinical practice that people who take ticagrelor have a lot of little nuisance bleeds,” said Wykrzykowska. “Even if they have STEMI or NSTEMI and indications for 1-year of treatment with aspirin and ticagrelor, these studies have shown that it’s okay after a month to switch down to aspirin and Plavix.”
In addition, several ongoing trials will provide important answers with regard to DAPT duration and stent technology, said Wykrzykowska.
Clinical Cardiology and Prevention
Secondary prevention of CVD saw some of the biggest trials in 2017, including FOURIER and CANTOS. “These results were more robust compared with 2015’s IMPROVE-IT trial that used the cholesterol absorption inhibitor ezetimibe (Zetia, Merck/Schering-Plough),” Michael Miller, MD (University of Maryland Medical School, Baltimore, MD), told TCTMD.
With regard to FOURIER, Miller said one aspect of the study that particularly impressed him was the continued benefit of evolocumab (Repatha; Amgen) when on-treatment LDL cholesterol levels were reduced to as low as 10 mg/dL. At these extremely low levels, there was no signal of risk observed, he noted. Alternatively, CANTOS demonstrated support for the inflammatory hypothesis and represented a new means of treatment outside of lowering LDL cholesterol.
Foy, on the other hand, wasn’t as impressed with FOURIER. In fact, what struck him the most was the hubristic thought that adding evolocumab to optimized high-dose statin therapy could abolish the residual risk of cardiovascular events in a secondary-prevention population.
“Sometimes people joke with me that I’m a medical Luddite,” said Foy. “It’s not that I’m against progress, because I’m not, but this study is interesting because evolocumab, more or less, wipes out LDL cholesterol, or at least to very low levels in patients with atherosclerotic cardiovascular disease. Even though the trial was positive, what this shows is that we didn’t do very much.”
Foy said that even though LDL cholesterol levels were reduced to a median of 30 mg/dL in the trial, more than more than one in 10 patients in both groups experienced the primary efficacy endpoint.
“As a society, do we want to continue to invest heavily in treatment of disease once it already occurs?” asked Foy. “As this trial shows to some extent, we may not have much further to go. . . . Should we declare victory from a medical standpoint and start thinking about how to invest as a society in disease prevention, which gets us away from the doctor’s office and puts resources back into the community?”
While there was a lot of exuberance over FOURIER, investment in primary prevention, with walkable cities and cycling infrastructure, for example, would likely go a lot further than spending billions of dollars for an additional therapy in patients with cardiovascular disease, suggested Foy.
To TCTMD, Khurram Nasir, MD (Baptist Health South Florida, Miami), also cited the FOURIER trial, and like Foy argued that hard decisions need to be made about how much payers are willing to spend for an expensive medical therapy that yields solidly effective, but not miraculous, gains. “The benefit will seem substantial to some and modest to others,” said Nasir. “However, it reignited the debate on issues related to its pricing, cost, and value.”
The American Heart Association (AHA), the American College of Cardiology (ACC), and other societies can play leadership roles to resolve cost-related issues, he added. “Moving forward, rather than debate whether a PCSK9 inhibitor is or is not affordable, we need a conversation about whether it represents value for money,” said Nasir.
Several experts also pointed to the importance of the new hypertension guidelines from the AHA, ACC, and nine other partnering organizations that redefined hypertension as starting at a blood pressure of 130/80 mm Hg.
“The idea that we should be more aggressive about treating hypertension would be a big change in terms of getting a lot more patients on therapy,” Anne Curtis, MD (University at Buffalo, NY), told TCTMD.
She said she’d still be wary of aiming for aggressively low blood pressure goals in older patients, and wouldn’t, for example, try to get an 85-year-old’s readings down to 120/80 mm Hg. “The reason I say that is that a lot of times they have pretty stiff vessels and once you start trying to get too aggressive in lowering the blood pressure you can have problems with orthostatic hypotension or getting them dizzy.”
Stephen Little, MD (DeBakey Heart & Vascular Center, Houston, TX), also told TCTMD that he was happy to see the renewed antisodium message that came along with the revised guidelines because it “really sort of doubles down” on what physicians in the past have advocated.
“Every month or so we hear about new pharmaceuticals to [lower blood pressure], but it’s nice to see that people have gone back to the basics,” Little said. “It’s not particularly sexy, but in terms of clinical utility, it’s easy . . . not to burden patients with insurance or economic challenges with new therapies” and instead simply place them on a low-salt diet.
Curtis told TCTMD that the “coolest thing” this year in the realm of heart rhythm is a small study looking at noninvasive cardiac radiation for ablation of ventricular tachycardia. Though the study included only five patients who had failed other treatments, the approach provided “amazingly good results” by reducing episodes of ventricular tachycardia by 99.9%, Curtis said. “It potentially has a huge amount of promise—it’s going to require a lot more work and they’re doing prospective trials on it—but . . . if you could stop having to use catheters to push around the heart to figure out where to fix these things, it would just be a revolution in the management of these arrhythmias.”
The CASTLE-AF trial was another important development in electrophysiology, said Curtis. The trial randomized patients with heart failure and A-fib to either conventional drug treatment or catheter ablation. The primary endpoint of all-cause death or unplanned hospitalization for worsening heart failure strongly favored the ablation arm.
“There seems to be increasing evidence that when patients have A-fib and heart failure, if you can fix the A-fib, you can improve outcomes,” Curtis said, adding that the trial could have an impact on practice by making clinicians choose ablation earlier in that patient subset.
And finally, Curtis pointed to studies of His-bundle pacing as a major development. With permanent pacemakers, the lead typically goes in the right ventricle, requiring the impulse to travel through myocardium. His-bundle pacing, however, uses a very small lead placed on the septum to recruit the His-Purkinje system to pace the ventricles. That approach has yielded success rates of 85% to 90%, although information on longer-term outcomes is still needed, Curtis said.
Structural Heart Disease
Ted Feldman, MD (NorthShore University HealthSystem, Evanston, IL), said the single biggest thing to move the dial in 2017 is the New England Journal of Medicine’s decision to publish, in a single issue, three randomized trials supporting PFO closure for stroke prevention. “After more than a decade of struggling with trying to convince the noninvasive world that PFO closure prevents recurrent cryptogenic stroke, there’s now no ambiguity. The three trials, RESPECT, CLOSE, and REDUCE, represent significant advances for the field, and most importantly those advances will have a positive impact on patients,” he said.
Mayra Guerrero, MD (NorthShore University HealthSystem), similarly placed PFO closure among the top news. “It really got our attention, but it did not surprise me,” she said. “Many of us knew that this would be positive and would just take time.”
Five-year findings from PREVAIL and PROTECT AF on the Watchman LAA closure device (Boston Scientific) also were a “highlight,” Guerrero observed.
Lars G. Svensson, MD, PhD (Cleveland Clinic, OH), told TCTMD that, for his center, the “most striking thing” is performing their first percutaneous tricuspid valve replacement. “We’ve done a number of patients now under compassionate use, and we’re now working with the [US Food and Drug Administration (FDA)] on a feasibility trial,” he said, adding, “That’s the big one for us.”
Moreover, “of course there’s a tremendous number of various devices that have popped up this last year both for the mitral valve and the tricuspid valve,” Svensson noted. “That’s a very exciting area of growth with transcatheter devices over time.”
An incremental advance that continued to be felt this year, Feldman agreed, is the growing momentum of transcatheter mitral valve replacement (TMVR). “We have seen and continue to see small strides being made by a variety of technologies to make the procedures more reproducible, and more often transseptal rather than transapical,” he said. Improvements in imaging have been critical here, he added. “With every endeavor we’ve ever had, imaging has led the way because you can’t go where you can’t see. We’re understanding how to look at those scans better and evaluate all the complexities of the mitral apparatus.”
Guerrero, too, cited early feasibility trials of TMVR, including her own MITRAL study, as well as results with the Tendyne and Intrepid valves. “Compared with other years, I think the common theme is that the findings of the recent trials [show better outcomes] than before with prior devices or prior attempts,” she said, echoing Feldman’s idea that “transseptal is a better way to go whenever possible” in mitral cases.
One milestone, Guerrero said, was the expanded FDA indications for Sapien 3 (Edwards Lifesciences), permitting its use in in mitral valve-in-valve procedures.
Another intriguing development, Svensson said, is the potential to combine multiple transcatheter devices in the same procedure. For example, the WATCH-TAVRmultinational study, just getting underway, is pairing the Watchman device with TAVR for patients who have both A-fib and aortic stenosis. Until recently, it was impossible to formally test these options, Svensson explained, because more than one investigational device cannot be combined in a single trial. Now, as more devices become FDA approved, it’s possible to study the effects of adding a newer contender to an existing therapy, he said.
Regarding the TAVR space, Guerrero said while there was no earth-shattering news, the accumulating trial data on lower-risk patients and initial forays into treating asymptomatic patients has kept the field’s interest. Specifically, the ongoing EARLY TAVR trial is changing clinicians’ perspectives, she commented. “We no longer have to wait for symptoms. We can take those patients with severe aortic stenosis and talk to them about early TAVR” with the hope of easier recovery and lower risk than surgery.
In the realm of cardiovascular imaging, Little told TCTMD that one of the most interesting topics explored in 2017 is the connection between cardiac amyloidosis and aortic stenosis as viewed on a new cardiac nuclear study called technetium pyrophosphate. “The gist of it is that there’s this phenotype of patients that we see with aortic stenosis that we send for TAVR and they have small ventricles, thick hearts, and a lot of diastolic dysfunction and it always gets ascribed to being elderly and having aortic stenosis,” he explained.
But studies this year have shown that amyloidosis is “very common” in those with aortic stenosis, who are often sent to undergo TAVR. At the moment, the data are merely “thought provoking,” Little said, but it’s likely going to lead to a more research given the lack of knowledge regarding the appropriateness of TAVR for these patients.
“One of the hardest things we do in the TAVR environment is deciding who’s not going to benefit from the therapy, but if somebody clearly has a burden of cardiac amyloid, then a TAVR is not going to fix that in any way,” he said. “Maybe there should be a discussion around futility and sort of potentially improving patient selection.”
Kevin Harris, MD (Minneapolis Heart Institute Foundation, MN), on the other hand, cited a very recent study that identified myocardial fibrosis in competitive male triathletes. In that study, the scarring detected by late gadolinium enhancement by cardiac magnetic resonance imaging was particularly pronounced in athletes who participated in long-distance events, such as Ironman triathlons.
Additionally, Harris pointed to a recent study that highlighted a subset of patients with hypertrophic cardiomyopathy (HCM) who have left ventricular apical aneurysms.
“It’s an interesting paper and gives us some good insights into a subset of patients with hypertrophic cardiomyopathy who are at a higher than expected risk [for arrhythmic sudden death],” said Harris. Additionally, HCM patients with left ventricular apical aneurysms were also at a higher risk of thromboembolic events.
Finally, Harris also cited a CT imaging study of patients with uncomplicated acute aortic dissection. In that study, imaging-based morphological features were combined into a prediction model to identify patients at high risk for late adverse events after an uncomplicated type B aortic dissection.
“The biggest trial of the year by far in my opinion was the recently-published ATTRACT trial, which did not hit its primary endpoint,” said Michael R. Jaff, DO (Newton-Wellesley Hospital, Boston, MA). “This was a highly anticipated trial that had been ongoing for many years and it’s a highly controversial area of therapy.”
Jaff also pointed to 12-month results of the ILLUMENATEtrial. The low-dose paclitaxel-coated Stellarex balloon (Spectranetics) showed superior safety and efficacy compared with angioplasty alone in a multicenter trial of 300 PAD patients with claudication. Stellarex “became the third to the market as a result of this trial,” Jaff noted. “Overall, it was a very impressive time course from invention to FDA approval.”
Finally, Jaff said the finding that carotid stent fractures are not associated with adverse events, as reported in the ACT I trial, was another important advance in endovascular medicine. “Everybody kind of believed that if you have a stent fracture it results in a higher risk of stroke and death and need for repeat interventions. But it turned out that none of those were the case,” he remarked.
Among the big news in heart failure this year, Paul Hauptman, MD (Saint Louis University School of Medicine, MO), pointed to the emergence of antidiabetic medications as therapies for reducing cardiovascular risk. In August, liraglutide (Victoza; Novo Nordisk), a glucagon-like peptide-1 receptor agonist, received an indication for reducing risks of MI, stroke, and cardiovascular death in adults with type 2 diabetes and established cardiovascular disease. That follows a similar decision for empagliflozin (Jardiance; Boehringer Ingelheim/Lilly), a sodium glucose cotransporter-2 inhibitor thatreceived an indication at the end of 2016 for reducing the risk of cardiovascular death in adults with type 2 diabetes and established cardiovascular disease.
“It used to be thought of as maybe it’s just an issue of safety, but now it looks like . . . some of these drugs actually have favorable effects on the heart,” Hauptman told TCTMD, noting that there has been a resurgence of interest in diabetes in the heart failure community.
He pointed out that two new trials were launched in 2017 to look at use of empagliflozin in patients with heart failure and either preserved ejection fraction (EMPEROR-Preserved) or reduced ejection fraction (EMPEROR-Reduced); patients are not required to have diabetes. “It’s a first attempt now to extend findings from a main diabetes study to see whether or not—in this particular case—empagliflozin could actually be a heart failure drug in addition to being a diabetes drug,” Hauptman said.
Other major developments, he added, were the approval of the HeartMate 3 left ventricular assist device (LVAD), the expanded destination therapy indication for the HeartWare LVAD, promising early study results for a novel drug for symptomatic hypertrophic obstructive cardiomyopathy from MyoKardia and an interarterial shuntfrom Corvia Medical, and continued refinements of heart failure guidelines.
Policy and Practice
For Frederick Masoudi, MD (University of Colorado Anschutz Medical Campus, Aurora), the biggest news of the year affecting everyone from physicians to patients, and hospitals to insurance companies has been “the fight over the Affordable Care Act,” or ACA. “It seemed like the ACA had nine lives at least at one point, although I think that’s probably changed fairly substantially with the new tax legislation that’s been approved by both the House and the Senate that eliminates the mandates as part of the ACA.”
Looking back over 2017, he said there were several times when “things were looking pretty good for the ACA despite a lot of attempts to undermine enrollment.” However, the “poor public understanding” of the reasons why it is necessary to maintain both coverage for preexisting conditions as well as mandates and subsidies has contributed to the success of the tax legislation, Masoudi commented, adding, “Certainly what’s happened here at the end of the year is I think bad news for the ACA.”
To TCTMD, Christopher Meduri, MD (Piedmont Heart Institute, Atlanta, GA), said one of the overarching themes of 2017 has been the growth of early feasibility studies in the United States.
“If I look back to just a few years, it really seemed like every US interventionalist at any major meeting was watching the revolution in valvular heart disease from the sidelines,” he said. “We always dreamed of being up there doing some of these cases, or being involved with the new technologies. Fortunately, not just for investigators, but also for patients, the FDA has really developed a collaborative atmosphere between sponsors and the sites doing the early feasibility work. Now, more and more technologies are being tested in the US.”
At TCT 2017, for example, there were presentations on several US-based, early feasibility valve-related trials, said Meduri. At his center, he has participated in two early feasibility studies, and at least three more valve-related technologies will be tested in the coming months. His institution has also been invited to participate in the FDA’s early feasibility trial network.
“I remember being a fellow and thinking, ‘You have to go to Europe to do anything.’ So I went to Europe for several months, trying to get my hands on some of these things I’d never get to do otherwise,” said Meduri. “Now, at some of the same places I trained at in Europe, there’s interest in coming over here [to the US] to watch us do things instead. It’s pretty crazy.”
This story was a collaboration by Caitlin E. Cox, L.A. McKeown, Michael O’Riordan, Todd Neale, and Yael L. Maxwell. Did we miss something? Let us know in the Comments section below what we left out, or have your say on Twitter using the hashtag #TopCardio2017.
Jeremias reports receiving institutional support from Philips/Volcano and Abbott Vascular and consulting for Philips/Volcano, Abbott Vascular, Boston Scientific, and Opsens.
Jang reports receiving educational grants and consulting fees from Abbott.
Wykrzykowska reports receiving grants/research support from Abbott Vascular and consulting for Abbott Vascular and St. Jude Medical.
Nasir reports serving on the advisory board for Quest Diagnostics and consulting for Regeneron.
Curtis reports receiving speaking honoraria from and serving on a data safety monitoring board for Medtronic and serving on the medical advisory board and adjudicating clinical trial results for Abbott (St. Jude Medical).
Masoudi reports serving as the chief science officer of the National Cardiovascular Data Registry. Guerrero reports receiving research grant support from Edwards Lifesciences, serving as a consultant/speaker for Abbott, and serving on the speaker’s bureau for Boston Scientific.
Svensson reports being an unpaid member of the executive committee for PARTNER trials I and II, chairman of the PARTNER publication committee, and an unpaid member of the executive committee of the COMMENCE trial, sponsored by Edwards Lifesciences.
Feldman reports consulting for Abbott, Boston Scientific, Edwards Lifesciences, and Gore.
Jaff reports consulting for Micell, Primacea, Vactronix, Venarum, and Volcano/Philips.
Foy, Miller, Little, Harris, and Meduri report no relevant conflicts of interest.
[dropcap]H[/dropcap]ardin County’s former EMS director has filed an anti-discrimination suit claiming he was wrongfully discharged from his job.
John Malcomson, a Radcliff resident, was suspended in June and discharged in July about one year after returning to work following a health emergency, which left him with a disability requiring some work-related restrictions.
In the four-page lawsuit filed last week in Hardin Circuit Court, Malcomson names Hardin Fiscal Court and Judge-Executive Harry Berry as defendants
Malcolmson asks the court for compensatory damages, reimbursement of legal fees and “any other relief to which he may otherwise be properly entitled.” No dollar figure is mentioned in the suit.
When the holiday weekend began Friday, defendants had not yet been served with the suit. Regardless, Berry said the county government would maintain its practice of declining comment on pending litigation.
During a public meeting in which Fiscal Court approved Malcomson’s firing, Berry said his recommendation stemmed from “misbehavior and inappropriate activity within the department.” Berry also said low morale and high turnover influenced his decision.
“Mr. Malcomson’s focus on the operations and business of the department is lacking. His availability is limited and unpredictable,” Berry said. “He routinely misses meetings, fails to meet expenses, demonstrates serious mood swings and is frequently disrespectful in his tone and demeanor toward his superiors and his subordinates.”
Malcolmson became emergency medical services director in August of 2014. As a result of an aortic dissection suffered in January 2014, he missed nearly 18 months of work. A complication required a partial left foot amputation and, according to the lawsuit, he was regarded as disabled by county government upon returning to work.
The lawsuit described a reprimand presented by Bryce Shumate, his supervisor, and Deputy Judge-Executive Jim Roberts following Malcolmson’s return to work. He was cited for failing to provide documentation of employee training. He claims in the suit the documentation had not been required during his initial two years of employment.
Malcomson, who missed work again in May 2017 with further complications related to the aortic dissection, said he was notified of his suspension June 22 and told by Berry he “was not capable of performing his job responsibilities.”
A civil suit provides only one side of a dispute. The county will file a formal response before the case proceeds in Hardin Circuit Court.
[dropcap]I[/dropcap] wanted to give everyone an update on me. Well, first off today is my wife’s birthday. So, we are going out to dinner tonight and watching at band. My son is home from his first semester at WSU. As well as my wife is going to pick up my daughter down at Greenlake -where she lives above PC Markets. We will have the entire family back together for the holidays.
I am a bit over 2 weeks now recovering from my broken ribs (8 and 9). I was goKarting and hit the wall on the right side and I think my ribs took the impact from the seat. Needless to say, I haven’t been doing anything lately. I am going to try and hit some tennis balls tomorrow with a guy on my USTA team. Our tennis season starts 1/7/18 and hopefully it will be another fun year. I am also going to play on an 18 and over team too! Plus I will continue to see how high I can get my ranking in singles in the 55’s division.
It will be 1 year on my new left hip come 2/17/2018. I am also going to be 2 years since my Prostate Cancer on 8/25/2018 and MOST important, I will be 15 years since my AD on 8/22/2018.
I need to really concentrate on my blood pressure as I still continue to have it. And, I need to do a better job of taking my meds daily. I still managed to take my Lexapro 20mgs.
It’s funny I am watching Elf for the 4th time and I just love this movie. I also like the Grinch with Jim Carey. I also just watched an interesting racing documentary called Senna. It was very well done, and had a sad ending. But.. fantastic story about racing.
In summary, I hope 2018 brings each and everyone of you the best year ever. We have so much to be thankful for. Family, friends, jobs, pets, and most importantly our faith.
[dropcap]A[/dropcap]fter an Horry County police officer was on duty patrolling last month, he suffered a traumatic heart injury and was rushed to a nearby hospital for life-saving surgery.
Now, 36-year-old KC Canterbury is home for recovery, but still faces a mound of medical bills as he heals.
“He’s one of those people that would literally do absolutely anything for anybody at any hour of the day,” said John Brantly, vice president of the Coastal Carolina Fraternal Order of Police Lodge #12. “He’s a very family oriented person, regardless of whether it’s blood family or blue family, you couldn’t find a better person.”
On the night of Nov. 25, Canterbury suffered from an aortic dissection and an aneurysm.
An aortic dissection is a serious condition in which the inner layer of the aorta, the large blood vessel branching off the heart, tears, according to Mayo Clinic. Blood surges through the tear, which causes the inner and middle layers of the aorta to separate — or dissect, hence the name.
An aneurysm is an “abnormal bulge or ballooning in the wall of a blood vessel,” Mayo Clinic explains. An aneurysm can rupture, cause internal bleeding and lead to death.
Canterbury was in critical condition when he was rushed to the hospital that night before he underwent an intensive surgery. He was released from the hospital on Dec. 11.
“It’s going to be a long road of recovery for him,” Brantly said. “That procedure takes an extreme toll on the body.”
Brantly started a YouCaring page to raise money for Canterbury’s expensive medical bills. As of Dec. 22, community members have raised $12,173 out of the $20,000 goal.
“Speaking for his family, they ask for prayers and support for his full recovery and the response from the community so far, has been overwhelming and nothing but positive, they greatly appreciate that,” Brantly added.
Canterbury has been in law enforcement for around 12 years and is the president of the Coastal Carolina FOP Lodge #12.
Brantly said checks are also being accepted, which you can pay to the order of SC FOP Lodge 12 and in the memo section write “KC Relief.” Checks can be mailed to the following address: S.C. FOP 12 P.O. Box 8455, Myrtle Beach, S.C., 29578.
[dropcap]R[/dropcap]ight side chest pain is a commonly reported symptom experienced by many. While it may not have the same connotations as left-sided chest pain, which is often related to having a heart attack, right-sided chest pain can also be associated with several relatively unappreciated consequences.
The left side of the chest is often given more attention due to the fact that the heart resides more so on this side of the body. However, serious health problems can often present with right-sided chest pain that shouldn’t be neglected.
Anatomy of right side chest pain
The chest or thorax region of the upper body has a number of important organs that reside within it that may present with chest pain if they become compromised in some way.
In particular, the right side of the chest is home to several structures including the right side of the heart, the three lobes of the right lung, the ascending aorta, the pulmonary blood vessels, and the esophagus. All of these are interlaced with a rich and diverse network of lymph nodes and nerve fibers.
Occurs due to inflammation of the cartilage connecting the ribs and the sternum (breastbone). Pain in this area of the chest can become so severe that it may mimic that of a heart attack or other heart-related condition. Treatment is focused on reducing pain until the condition resolves on its own.
2. Muscle strain
Also known as a pulled muscle whereby a muscle has become overstretched or torn. Often times, a muscle strain is the result of fatigue, overuse, or improper use of said muscle, most commonly involving the muscles of the lower back, neck, shoulder, and hamstring. Muscle strains can be a source of pain and decreased movement. Mild-to-moderate strains can be successfully treated with ice, heat, and anti-inflammatory medication, while more severe cases may require more intensive medical treatment.
3. Cervical or thoracic spine conditions
Conditions that result in the abnormality of the vertebral disc of the spinal column, such as disc disease or a compressed vertebra, may cause pain to be referred to the right side of the chest. Metastatic cancer of the cervical spine may also spread to other areas in the thoracic cavity lending to chest pain on the right side.
Heart and blood vessel causes:
1. Coronary artery disease
Due to hardening or narrowing of the blood vessels of the heart and is primarily due to the build-up of cholesterol and plaque in the inner walls of these vessels, known as atherosclerosis. As this condition progresses, less and less oxygenated blood is able to reach the heart, leading to chest pain (angina) or even a heart attack. Coronary heart disease is the most common type of heart disease and the leading cause of death for both men and women in the United States.
Refers to the swelling and irritation of the sac-like layer of the heart called the pericardium. Inflammation of this membrane often causes chest pain when the irritated layers of the pericardium rub against each other. It is often difficult to determine the cause of pericarditis in the majority of cases; however, viral causes are often suspected.
3. Dissecting aortic aneurysm
A very serious yet relatively uncommon condition involving the large blood vessel of the heart. It is characterized by the inner layers of the aorta tearing apart from the outer layer, creating a pocket where blood can collect. This leads to further dissection of the two layers. If the blood-filled pocket ruptures, it can be fatal unless emergency medical treatment is provided. Aortic dissection is most frequently seen in men in their 60s and 70s.
Lung related causes:
1. Pulmonary embolism
Occurs due to blockage of one of the pulmonary arteries in the lungs. Pulmonary embolisms are due to free-floating blood clots, called emboli, that dislodge from another part of the body, often from the lower extremities after a condition called deep vein thrombosis. The blockage of blood flow to the lung can be life-threatening and should be treated right away to improve the chances of survival.
2. Lung cancer
Developing a tumor of the right lung or near the lymph nodes can cause pain on the right side of the chest. Pain caused by lung cancer may also travel to the shoulder blades. Unfortunately, there is no screening test for the early diagnosis of lung cancer, resulting in most cases being far along in the course of the disease before they are identified. Other symptoms of lung cancer include shortness of breath and a persistent cough.
A serious and possibly life-threatening respiratory infection. It can be caused by viruses, bacteria, and fungi, resulting in the air sacs of the lungs filling with fluid or pus. It is estimated that about 900,000 Americans get pneumococcal pneumonia every year, and about five to seven percent of them die from it. Pneumonia can infect people of all ages, being most dangerous in those who are very young and very old, as their immune systems aren’t very strong. Symptoms of pneumonia often include coughing with phlegm or pus, chills, and difficulty breathing.
Refers to a collapsed lung that occurs when air leaks between the lung and the chest wall. As a result of this defect, air pushes on the outside of the lung and makes it collapse, allowing it to expand during the normal breathing process. A pneumothorax can be caused by a blunt or penetrating chest injury, certain medical procedures, or damage from underlying lung disease. Symptoms often include sudden chest pain and shortness of breath.
5. Pleural effusion
Occurs due to the buildup of fluid in the pleural space of the lungs, which is the area between the layers of tissue that lines the lung and the chest cavity. The type of fluid residing between these layers can be very helpful in the diagnostic process. If there is transudate fluid, heart failure or cirrhosis is very likely. If the fluid is exudative, lung infection or malignancy may be the cause.
Also known as pleuritis, this condition refers to the inflammation of the pleura, a membrane consisting of a layer of tissue that lines the inner side of the chest cavity and the layer that surrounds the lungs. Symptoms are often described as sharp chest pain that worsens with breathing.
Digestive tract causes:
1. Gastroesophageal reflux disease (GERD)
Perhaps the most common cause of epigastric pain (pain felt in the upper chest), GERD leads to a burning sensation behind the sternum that is felt in the epigastric region. GERD occurs due to the regurgitation of stomach acids back up into the esophagus, leading to tissue irritation. GERD can be managed with lifestyle changes and the use of over-the-counter antacid mediation. However, some individuals may need stronger medication or even surgery to remedy their symptoms.
2. Esophageal spasm
Painful contractions of the esophagus can feel like sudden, severe chest pain that can last for a few minutes to a few hours. While in the majority of cases, treatment may not be required, sometimes spasms are frequent enough to prevent the passage of food and liquids from traveling down the esophagus.
Inflammation of the gallbladder typically caused by gallstones blocking the ducts that exit the gallbladder itself. Other potential causes of cholecystitis include bile duct problems and tumors leading to obstruction of bile flow. Symptoms often present as severe pain in the right abdomen, pain in the right shoulder or back, nausea, vomiting, and fever.
Also known as biliary calculi, these stones are formed due to imbalances in the level of cholesterol in the body. However, they may also form if the gallbladder does not empty efficiently. Gallstones may range in size from one millimeter to several millimeters and usually do not cause any problems until they are big enough to block the bile ducts, which lead to pain. Symptoms of gallstones present as severe abdominal pain, jaundice (yellowing of the skin and eyes), and fever.
A condition whereby inflammation has overtaken the pancreas, leading this organ to not function as it should. The pancreas is an organ of the body that produces enzymes that help with digestion as well as hormones to regulate how the body processes sugar. There are a number of different causes of pancreatitis, such as alcoholism and gallstones.
6. Peptic ulcer
Due to ulcer formation inside the lining of the stomach as well at the upper portion of the small intestine. Peptic ulcers often lead to pain that radiates up to the epigastric area. Ulcers can be the result of H. Pylori infection, long-term aspirin use, or the use of other painkillers such as ibuprofen and naproxen. Contrary to popular belief, peptic ulcers are not caused by stress or spicy foods, but can make the symptoms of an existing ulcer worse.
7. Liver cirrhosis
A late stage complication of liver disease that is characterized by scarring of the liver (fibrosis). The condition prevents this important organ from performing necessary functions, such as detoxifying harmful substances, cleaning the blood, and making vital nutrients. It can be caused by several diseases such as hepatitis and chronic alcoholism.
What are the symptoms of right side chest pain?
Depending on the underlying cause, right chest pain may also be accompanied by the following symptoms:
Feelings of pressure, squeezing, or fullness in the chest
Severe shooting pain in the arms, back, neck, jaw, or stomach
Treating your specific cause of right side chest pain will depend on the root cause of it. Some conditions may only require lifestyle changes and diet modification, while others require lifelong use of medications or even surgery. It is important to work closely with your doctor to find out all the things you can do on your own to reduce the risk of serious complications.
Should you worry about right side chest pain?
There are many causes that lead to the development of right sided chest pain. However, most cases are not directly related to the health of your heart. If you feel chest pain on your right side, it is relatively safe to assume that it is not caused by your heart and that another cause is most likely the culprit.
However, if you are experiencing extreme chest pain, regardless of the location, going to your local emergency room is highly recommended.
[dropcap]C[/dropcap]elebrities, children and families from Essex are making a charity’s celebrity Christmas campaign viral – and are now calling on grime and hip hop superstar Stormzy to get involved.
Heart Research UK’s ‘Sing for you Heart’ sees members of the public and celebrities sing, donate and nominate – by uploading a short video of themselves singing on social media, texting SING to 70144 to donate £3 and then nominating up to three others.
The money raised will help further fund pioneering medical research into heart disease.
The viral campaign has reached over 12 million people online and seen support from Essex author and blogger Giovanna Fletcher (wife of McFly’s Tom Fletcher), Irish superstars Boyzone, Strictly Come Dancing star Brian Conley and Davood Ghadami, Britain’s Got Talent’s Alesha Dixon, Loose Women Linda Robson and Stacey Solomon, panto favourite Christopher Biggins, and ITV’s Good Morning Britain’s Piers Morgan.
For Heart Research UK Ambassador and Thurrock’s Vikki Renoldi-King, Sing for your Heart is more personal.
Vikki’s brother Julian died in 2014 from aortic dissection, a rare condition where the inner layer of the aorta tears. Julian, a father of three, was only 44.
Now the 40-year-old from Linford, who has raised £5,000 for Heart Research UK in the past by completing sponsored walks around Essex, wants the rest of the country to follow the county’s lead.
Vikki said: “Julian loved to play guitar and listen to music. When he was alive he would often send me songs to listen to and after he died I played his music often to feel close to him.
“Sing for your Heart is a special campaign, so to get friends and family involved to sing to raise money for heart disease just makes it all the more personal to us”.
One member of the family got extra special support from her friends – 11-year old Annie Renoldi-Ludlow from Rectory Road, West Tilbury. Annie performed her Sing for your Heart video with her Youth Creations band, Young Vibes from Stanford-le-Hope. The group, made up of 7-11 year olds performed Stormzy’s Blinded By Your Grace.
Annie said: “We love singing, especially Stormzy songs. We had so much fun. If Stormzy heard it and did his Sing for your Heart, it would be amazing.”
Vikki added: “I’m asking all of Essex to share Young Vibes’ video, tweet @Stormzy1 with #STORMZYsingforyourheart and donate £3.
In order to prevail in a medical malpractice action, the plaintiff must show, by a preponderance of evidence standard, that the defendant owed a duty of care to the plaintiff, the defendant was negligent in providing that care, the plaintiff was injured, and the legal cause of the injuries was the negligent care. All four of these elements must be shown for the plaintiff to win.
The most compelling type of medical malpractice case is the failure to diagnose a life-threatening condition which results in the death of the patient. Myocardial infarction, aortic dissection, certain malignancies, and neurologic injuries represent these types of cases. If the provider can show that he considered these types of conditions and he took steps to make the diagnosis, then he will likely win even if the diagnosis is missed.
There are false positives and false negatives for all diagnostic tests and the courts recognize this. In general, expert testimony is needed to confirm or deny that the provider did what a reasonable provider would do if faced with the same or similar circumstances. Expert testimony is required because most people do not have the knowledge or experience needed to make decisions on standards of care by themselves. Under the law, an expert can be used to educate the trier-of-fact to better understand the evidence or determine a fact in issue.
In most states and in the federal courts, an expert witness must be qualified by knowledge, skill, experience, training, or education so that he can testify in the form of an opinion or otherwise. Before an expert is allowed to testify, the court must act as a gatekeeper to determine if the expert’s testimony will substantially assist the trier-of-fact. The factors that the court will use in making this determination are (1) whether the scientific evidence has been tested, (2) whether the evidence has been subjected to peer review and publication, (3) whether a potential rate of error of the evidence is known, (4) whether the evidence is generally accepted in the appropriate scientific community, and (5) whether the expert’s research in the applicable field has been conducted independent of the litigation. These factors come from the Daubert v. Merrell Dow Pharmaceuticals, Inc. 509 U.S. 579 (1994) holdings which relate to what an expert witness is allowed to testify about in a malpractice action.
Prior to Daubert, the federal courts used the “generally accepted” standard which originated from Frye v. United States 293 F. 1013 (D.C. Cir. 1923). In Frye, the court held that novel scientific testimony is admissible in federal courts if the testimony had been “generally accepted” in the appropriate scientific community where it had originated; expert testimony must be based on scientific methods that were reasonably used and accepted. Most states then came on board with this standard and it had been used for over 70 years.
Most states and the federal Courts have adopted the holdings of Daubert but some have not. There are still 13 states which follow the standard of Frye.The Frye standard is a much easier to meet than Daubert. The courts that use the “generally accepted” standard reason that the jury should be allowed to rule on the relevance of the testimony and there should be no prior screening by a judge.
The “standard of care” is defined by statute in most states. For example, in Tennessee, it is “the recognized standard of professional practice in the profession and the specialty thereof, if any, that the defendant practices in the community in which the defendant practices or in a similar community at the time of the alleged injury or wrongful act occurred.” Most states do not have the community standard element that is in the Tennessee statute.
In medical practice, there are many conditions which now have recommendations for care and treatment. These recommendations come from committees comprised of recognized experts in the field. I was at a lecture for continuing legal education where a well-known plaintiff’s attorney stated that, as far as he was concerned, the “best practices” paradigms recommended by these committees were now the “standards of care” and any health care provider who did not follow the paradigms was committing medical malpractice. He was wrong.
Most, if not all, of the publicized treatment recommendations have a disclaimer which recognizes that it is up to the treating clinician to decided on the proper care of a particular patient. The law has not changed; the standard of care is still the care that would be provided by a reasonable physician who is faced with the same or similar circumstances. If a reasonable clinician would not have followed the committee’s recommendations, then the standard of care may still have been met.
Here is an example where the paradigm may not be the standard of care. Under the committee recommendations, a person who presents to the hospital with an acute coronary syndrome should be given morphine, oxygen, aspirin and nitrates. According to the above mentioned plaintiff’s attorney, a person not getting all elements of this treatment regimen would be the victim of malpractice. However, physicians would not give morphine to a person with a low blood pressure or to someone who is allergic to the drug. A patient who has severe chronic obstructive pulmonary disease may stop breathing if given supplemental oxygen. A person who has gastrointestinal bleeding or is allergic to aspirin should not be given that drug. We know from TV commercials that a person on Viagra or Cialis should not take nitrates as that could lead to a sudden dangerous drop in blood pressure.
Opposing attorneys will try their best to discredit the expert witness on cross-examination and they have many weapons in their arsenal to accomplish this goal. One strategy is to let the jury know how much has already been paid to review the medical records, study deposition testimony and answers to interrogatories, and how much is being charged per hour to do the reviews, give testimony at depositions, and testify at trial. The amounts can be substantial and many professionals make a significant income performing as an expert witness. If it can be shown that a particular witness has been paid a lot of money, then the jury may conclude that his opinion is being bought; the credibility of the witness will suffer accordingly.
Although there are many other ways to attack an expert on cross-examination, one of the best is to show the jury that he may not be an expert at all. This can be done by exposing weaknesses in the expert’s curriculum vitae. A national survey of 2.6 million resumes done in 2003 showed that 41 percent lied about their work experience and 23 percent lied about their credentials or licenses. A surprising 41 percent lied about their education (Thomas A. Buckhoff, Preventing Fraud by Conducting Background Checks, CPA J., Nov. 2003). Opposing attorneys now routinely check the backgrounds of medical expert witnesses and they will even look at social media sites to see if they can find some incriminating information that can be used to attack their credibility.
If it can be shown that the medical expert was willing to embellish on his qualifications, it is logical to assume he has embellished his medical opinions, also.
There are few trial lawyers that can show superior knowledge to the expert in the medical field under discussion. After all, they are lawyers and it is unlikely they have ever treated an actual patient. Book knowledge is just not enough to be a good physician and that is probably why it is called the “practice of medicine.” By practicing, you are hopefully improving your knowledge and skill as a physician all the time.
If the physician can show he used his best clinical judgment at all times while caring for the patient, he will likely win his case even if the results were not good. Physicians are forced to make judgments, some of which do not work out well for the patient. These decisions are made with the best data available; they cannot rule out every single diagnosis or provide perfect care. The courts and the juries recognize this and will generally rule in favor of the physician who has used his best judgment and done it in a timely fashion. The physician can be his own best medical expert witness in this regard.
Dr. Weiman is the author of two books, Medical Malpractice and Fundamental Issues in Health Care Law.
Dr. Weiman’s website is www.medicalmalpracticeandthelaw.com
I once cared for a patient in her 20s who came to the emergency room for a sore throat and a rash. While she was in no acute distress, she explained that her throat hurt and unusual bumps were erupting on her hands.
But then she mentioned starting a new antibiotic. My internal alarms sounded. Her symptoms were signs of a potentially life-threatening condition that required specialty services not available at my small hospital. I arranged for transfer to a tertiary care center. The next week I received a follow-up letter from her care team. No more than 36 hours after I saw her, she was in the intensive care unit, on a ventilator, fighting for her life.
The lesson is that even if not every rash and sore throat is a life-threatening condition, life-threatening conditions don’t always present themselves as obvious emergencies.
Anthem is justifying the move by pointing to significantly higher emergency room costs compared to non-emergent clinics, citing such so called “non-emergency” services as annual sports physicals, pregnancy tests and ingrown toenails. They argue this cost is inevitably passed on to consumers.
However, the list of Anthem’s non-emergent diagnoses does not stop there. Under this enforced policy, more ambiguous complaints like chest pain with breathing and bloody urine can also be deemed unworthy of an ER visit. The American College of Emergency Physicians has uncovered almost 2,000 of these non-emergent diagnoses in Missouri alone.
Hindsight is always 20/20. But patients don’t have the benefit of a medical evaluation and test results, nor the medical training to help them decide on their on whether their condition merits emergency treatment. And for that reason, the Prudent Layperson definition was added to the federal Emergency Medical Treatment and Labor Act (EMTALA) in 1994, defining an emergency condition as one that would lead a prudent layperson, with an average knowledge of medicine, to believe that his or her health is in jeopardy. It is the patient’s presenting symptoms, not the final diagnosis, that legally determines authorization and payment for emergency claims.
But even emergency physicians are not soothsayers. EMTALA mandates that no patients are turned away from the emergency room without a medical screening exam to rule out dangerous conditions. If a patient presents with chest pain, the bucket of possible diagnoses is overflowing, from benign ailments like muscle strain or heart burn, to life-threatening conditions like heart attack, pulmonary embolism or aortic dissection. Testing is often required to tease out the difference.
Anthem’s strategy of denying payments for these evaluations when certain non-emergent diagnoses are made all but ignores emergency physicians’ legal obligations under EMTALA. Regardless of Anthem’s policies, ER patients will continue to be evaluated. However, now, physicians and hospitals abiding by the law will be risking significant losses for legitimate services wherever it turns out patients cannot pay. More importantly, patients with genuine concerns face the risk of incurring a burdensome, unplanned, out-of-pocket expense. Some patients, fearing an enormous bill, may not seek help, potentially suffering dire consequences.
And, yes, amid these casualties, there may also be some reduction in non-emergent ER visits.
Rather than penalizing patients who believe their condition warrants an emergency visit, incentivizing them when they utilize appropriate services instead will likely also reduce non-emergent ER visits, and not deter patients from seeking appropriate emergency care when they should. By denying payments for certain non-emergent diagnoses determined after the fact, Anthem is merely pushing a healthcare cost hot potato off their lap and into the arms of legally obligated emergency care providers.
How many patients will suffer preventable injury or even death because they now fear a bill for a legitimate emergency department visit? That hindsight will be 20/20.
Dr. Cerundolo is a physician practicing emergency medicine in New Hampshire.
Regular physical activity is a common therapy for people with cardiovascular problems, but not recommended to those with Marfan syndrome, a rare disease of the connective tissue affecting the cardiovascular system. A study conducted jointly by the University of Barcelona and the August Pi i Sunyer Biomedical Research Institute (IDIBAPS) has analysed for the first time this medical recommendation, analysing the impact of exercise in the development of the disease in mice. The results, published in the scientific journal Journal of the American Heart Association, show that moderate exercise reduces the progression of aortic aneurysm, one of the most severe symptoms of the disease, which can cause arteries to break.
The study opens the door to studies on humans to verify the positive effects of this kind of exercise in patients with Marfan syndrome.
Hypothesis without experimental evidence
Physical activity increases the blood the heart pumps per minute. This increase has an impact on the aorta. “So far, it was thought that this mechanic impact, together with a discrete increase of the arterial pressure, could have negative effects on a weak arteria, like the case of patients with Marfan syndrome, and therefore it would dilate faster if the patient exercised,” says Gustavo Egea.
To test this hypothesis, researchers analysed the effects of moderate exercise on rice with Marfan disease. “During the five months of the experiment, the aorta of those mice who did not exercise dilated twice compared to those mice that were not ill. Mice which exercised experienced a reduction of this dilation until it was indistinguishable from those without the disease,” says Eduard Guash. Also, among the beneficial effects, researchers also saw that moderate exercise for five months reduced the size of the heart (cardiac hypertrophy) compared to those mice that did not exercise.
“The new results go against a clinical concept that was accepted without any experimental evidence to prove it: it said that all physical activity increased the risk of speeding up the aneurysm progression” says Gustavo Egea.
Thinking lifestyle recommendations twice
Although results in animals should be carefully interpreted before bringing them to patients, researchers highlight that the study provides experimental data that was not available before. “Our work opens the door to conducting studies on humans, but does not verify directly the non-selective recommendation of physical activity to patients with Marfan syndrome,” says Gustavo Egea.
Moreover, the researchers note that the study is focused on the impact of moderate exercise, which is the one recommended to people in general and which has been proved to bring cardiovascular benefits. “We did not face the effects of intense and long exercise and we cannot apply our results to other kinds of exercise –other than resistance,” says Eduard Guasch.
Marfan syndrome is a genetic disease caused by the mutation of a gene that codifies fibrilin I protein, one of the two main compounds of elastic fibers that make up the connective tissue. As a consequence of this mutation, the elastic fiber assembly in tissues is wrong, and therefore the function of relaxation is lost and tissues get damaged faster.
All tissues with elastic fibers or fibrilin I microfibriles are affected, like skin that gets stretch marks, or lungs with emphysema, and the eye lens that moves and causes blindness. The most significant one out of all these dysfunctions is the accelerated weakening of the ascending aorta, which leads to aortic aneurysm and the following dissection and breaking of the aorta.
These clinical manifestations are what is known as Marfan syndrome, which, despite being a minority disease, has a high prevalence of 1/5,000 patients and is hard to diagnose.
People with Marfan are tall with disproportionately large extremities. The average life of non-diagnosed people is around 40 years old, and around 50% of people with this disease are not diagnosed. In Catalonia, there can be around 1,500 people with it. The diagnostic is made with a study of clinical manifestation with a score. When in doubt or verification a genetic analysis can be carried out. Nowadays, the only effective therapeutic solution is surgery, and diagnose and regular monitoring of the aneurysm progression with imaging techniques are essential to increase the life of the patients.
More information: Aleksandra Mas‐Stachurska et al. Cardiovascular Benefits of Moderate Exercise Training in Marfan Syndrome: Insights From an Animal Model, Journal of the American Heart Association (2017). DOI: 10.1161/JAHA.117.006438
[dropcap]A[/dropcap]fter cheating death, time to enjoy our beloved Christmas Tournament.What I do know is, I should be dead. Two different cardiologists told me 80 percent of the time the aortic dissection I had would have been fatal.
Wikipedia defines an aortic dissection as “an injury to the innermost layer of the aorta which allows blood to flow between the layers of the aortic wall, forcing the layers apart.”
Let me tell you, you don’t want the layers of your aortic wall forced apart.
But with a lot of love from family and friends and a world-class doctor in Dr. Stanley Tam, I’m back.
For the first time in 42 years, I missed the highlight of my year last December, the annual Commonwealth Motors Christmas Classic. For my money, the premier boys basketball tourney in the state.
But it’s not going to happen again! I’ll be at Methuen High with bells on Dec. 23, 27 and 29.
Here is my story.
It was just about a year ago. I had returned from the Christmas Tourney banquet at DiBurro’s in Haverhill, written my story and proofread The Eagle-Tribune Monday sports pages.
I was heading home at about 2 a.m. the morning of Dec. 12. Suddenly, I wasn’t feeling well and/or my car broke down in an early-morning snow storm. It may have been both.
I woke up my wife, Yadira Betances Muldoon, and she was going to pick me up on the side of the road on Rte. 495 in Lawrence.
She sensed something was wrong and thankfully called the police. A kindhearted state trooper, I still don’t know his name, took good care of me and that was blessing No. 1 in a series of blessings that enable me to write this story.
MedFlight to Boston
The next thing I remember it was three days later. I was in intensive care at St. Elizabeth’s Hospital in Boston, where I had been MedFlighted that Monday morning from Holy Family Hospital in Methuen.
After an 18-hour emergency surgery, I now had a patched up aorta, a nasty 9-inch scar on my chest and too many tubes to count.
Facebook pronounced me well on the comeback trail. But the well-meaning friend couldn’t have been more wrong.
At about the time I was getting flooded with relieved compliments on social media, I was actually being rushed to a second 12-hour emergency surgery for internal bleeding.
Against doctors’ advice
Drip by drip I heard I was less than an ideal patient. I pray that was due to medication and the extraordinary situation I found myself in and not that I was once again being a jerk.
But when you are comatose for a few days, then semi-comatose, well, that’s my excuse.
I was pulling out all my tubes to the shock of my saintly nurses and wife. Hospital orderlies had to tie me to the bed.
One day I was frantically calling every number in my cellphone looking for a ride home to a comfortable bed, a stocked refrigerator, a computer with Wi-Fi and a clicker I had mastered.
Four things St. Elizabeth’s couldn’t offer.
I tried to bribe one colleague with $300 for a ride from Brighton to Methuen. Since I probably don’t have $300 to my name, that was pretty preposterous.
About 30 horrified people called my wife, “Um, Yadira, did you know Mike was planning on leaving the hospital against doctors’ advice.”
I still can’t believe that one, but I looked all sorts of doctors and administrators in the eye and said, in so many words, “Get me the %^&*# out of here.”
I actually told them, “I’m going home to sleep and will be back later.”
The fact they were some of the best medical minds in the world and I was a sportswriter who flunked Mr. Cayot’s sophomore biology class in high school, didn’t dawn on me.
Thankfully, enough relatives and friends convinced me I didn’t know what I was doing and all but restrained me.
If you have to die, you don’t want it to be because of Irish stubbornness.
But shortly before Christmas, I was released. I figured I’d dance an Irish jig and then take in the Christmas Tourney. Maybe cover the tourney for my 31st straight year.
Not so fast. I tried to walk down the hall at St. Elizabeth’s and nearly collapsed due to overwhelming fatigue.
No, I was bed-ridden for another month.
Defying long odds
I’m not a very introspective man. When I heard those chilling “80 percent dead” odds, my reaction was something like: “Wow, aren’t I lucky. Pass the clicker.”
My second reaction was that this will give me an excuse to stop dying my hair after 35 years.
But I am blessed to be here and I wonder if I would be here if it weren’t for my wife, Yadira, who moved into St. Elizabeth’s for 12 days and was there every step of the way.
I was hoping Yadira was with me in the helicopter ride to St. Elizabeth’s so that might count as a unique Christmas gift. She wasn’t, it didn’t. I’d better get her some Louis Vuitton red shoes this Christmas!
So many people to thank
I’m still working on some cool stories to explain the 9-inch vertical scar down my chest and two-inch horizontal one.
Maybe something like: “The Gronkowski brothers were acting up and I got right into Rob’s face …”
There are so many people who were so good to me. My wife, my mother, Joanne, my stepson Gabriel Martinez and my four siblings (Bobby, Kevin, John and Maura) certainly top the list.
All my Tribune sports colleagues were wonderful especially sports editor Bill Burt, a regular at St. Elizabeth’s, and Dave “Duffy” Dyer.
Even today, if I’m having a bad day I’ll go to the article Duffy wrote: “No. 1 Christmas wish: Full recovery for Mike.”
It’s an immediate pick-me-up.
My friend, former Andover basketball great Ted Kelley, hurried back from some far-flung locale thousands of miles away.
So he could be in my room for an hour while I was comatose!
St. E’s had a special nurse, and again I hate to say I don’t even know her name.
But when you have urine and feces on you, cramped in the bed at my height (6-foot-6) and frustrated beyond belief with no computer handy, a gifted and kind nurse can make all the difference.
Through the entire ordeal, and I’ve been just about 100 percent for 8-9 months now, this is what I was struck most by: People are so nice and so caring.
Other people in the newsroom I could never thank enough include photographer Carl Russo, community news editor Betsy Curry, who made a massive plate of her world-famous brownies when I returned, and our former night editor Joel Barrett.
Time to go to work
When a major storm hit, I panicked. I’ll be housebound for a week. I’ll have to miss work … on the heels of missing almost two months! The guys had already worked themselves to the bone carrying me and now this.
Muldoons are a lot of things, but we’re workers. I’ll never forget my the 50-something-year-old mother trudging three miles through 20 inches of snow to get to her job one time as a nurse at Phillips Academy.
And 40 years later, she has never mentioned it once.
But now I was going to be “that guy” everyone wanted to strangle.
So self-pity was setting in. Would I even be able to go back to my job which I enjoyed so much?
Then I hear the early-morning hum of a snowblower. It was my Methuen neighbor, PMA basketball coach Brian Martin, cleaning every nook and cranny of the driveway.
And he did it the entire winter, refusing to take a penny. What a guy.
Another huge boost came from Central Catholic. Raider athletes and coaches put together a 3-minute video, which was better choreographed than “Hamilton.”
I’ll never be able to thank them enough for it.
Love of Christmas Tourney
In addition to a life-and-death medical situation, taking enough drugs to put down an elephant, and battling what seemed to be depression, I so missed “The Christmas Tourney.”
The 42nd annual Commonwealth Motors Christmas Classic was right around the corner and for the first time ever I wouldn’t be there. Heck, I’d covered it for the last 31 years and it was always the highlight of my writing career.
No. 2? I couldn’t even answer that it was so far down the list.
Since my father is gone, I can admit it. I used to skip church to go to those early tournaments. It was hoops heaven.
I watched my buddies Ted Kelley and the Perry brothers become legends. I watched in awe and was determined to properly capture the greatness of Scott Hazelton and Carson Desrosiers and all those great Central Catholic teams.
I always wished I was as cool as Lawrence legends Rigoberto Nunez and Elinton Rosario.
This was my baby, my turn to shine. And it was taken away.
I might not know as much about sports as Hector Longo. Or have Bill Burt’s resume. Or Dave Dyer’s work ethic.
But dammit, I would leave no stone unturned so that you might get the occasional, “Hey, I really enjoyed that Christmas Tourney story you did on …”
So this year will be special. More special than people will ever know.
A 72-year-old woman presented to the hospital with 3 days of emesis, anorexia and generalized fatigue without any report of chest pain. Her physical examination was significant for mild confusion and bright red blood per rectum. On presentation she had bilateral lower extremity weakness, but was able to move both lower extremities; dorsalis pedis pulses were present bilaterally.
Initial laboratory examinations demonstrated a white cell count of 16.7 K/μL, creatinine of 3.0 mg/dL, aspartate aminotransferase/alanine aminotransferase of 1189/382 U/L, lactate of 4.5 mmol/L and a troponin of 0.52 ng/mL. ECG was unremarkable. An initial chest X-ray showed a dilated aortic arch and a modestly widened mediastinum.
A CT scan of the chest was not done. Several hours after presentation, the patient was noted to have flaccid lower extremities and diminished distal pulses, but with no skin changes or signs of distal ischaemia.
Duplex ultrasound of the lower extremities demonstrated biphasic waveforms of distal vessels with no areas of occlusion or evidence of embolisation. Transthoracic echocardiogram revealed a small pericardial effusion with grossly normal ventricular function; the aorta was not well visualised…….
CHICAGO, Dec. 15, 2017 /PRNewswire-USNewswire/ — Phillip Lonergan has hiked the Himalayas, the Alps, the Pyrenees and the Rockies, but an aortic aneurysm — a potentially deadly bulge in the largest artery in the body — threatened to be the toughest challenge of his life.
Northwestern Medicine Logo (PRNewsfoto/Northwestern Medicine)
Now, the Chicago-based graphic designer is on his way to exploring new trails after a first-in-Illinois procedure where Northwestern Medicine cardiac surgeon S. Chris Malaisrie, MD repaired and stabilized his damaged aorta with an investigational device known as a “frozen elephant trunk” graft because of its shape and stiff material.
The Thoraflex™ Hybrid Device is used to repair an aorta damaged from either an aortic aneurysm, or a bulge or ballooning in the aorta’s wall, or dissection, which is a tear in the aorta’s wall. The aorta runs from the heart through the chest and abdomen, carrying oxygen-rich blood to the body.
The frozen elephant trunk graft is designed to repair the damage in one surgery when previously, surgeons required two separate procedures to make the complicated repair.
“This device makes the procedure easier for the patient and reduces the amount of time the patient is on the heart-lung machine during surgery,” said Dr. Malaisrie, who is co-director of Northwestern Medicine’s Thoracic Aortic Surgery Program and an associate professor of surgery-cardiac surgery at Northwestern University Feinberg School of Medicine. “It’s a potential breakthrough in the surgical treatment of arterial disease, and we are pleased to be part of this clinical trial investigating its efficacy.”
Lonergan’s aortic bulge happened in 2010, and was repaired successfully at the time at Northwestern Memorial Hospital. However, the repair wasn’t permanent and in 2016 Dr. Malaisrie told him he qualified for a clinical trial investigating the frozen elephant trunk device.
Lonergan said he always knew he would need additional surgery on the aorta, and was pleased there was an investigational option.
“I’ve known this was coming for six years,” he said. “Everybody here at Northwestern Memorial Hospital has been great, from the surgeon and probably dozens of other staff who saved my life in 2010 to Dr. Malaisrie, who extended it and spared me an additional surgery. I looked at this clinical trial as my chance to give back to medicine.”
“This has helped me get back on my feet,” he added. “Now, I want to see more of the United States, and maybe travel to the Middle East.”
The Thoraflex™ Hybrid IDE study will enroll as many as 80 participants in 14 centers in the United States. Northwestern Medicine hopes to enroll 10 individuals. Dr. Malaisrie has performed nine surgeries as part of the trial.
The Bluhm Cardiovascular Institute is part of the Northwestern Medicine health system, with multiple sites of care in Chicago and the region. Northwestern Memorial Hospital currently is ranked first in in the United States for heart failure survival, second in the United States for the survival of stroke and in the top 10 for the survival of heart attack, the three most dire cardiovascular health threats. Northwestern Memorial Hospital’s heart and heart surgery program is ranked seventh nationally and first in Chicago, Illinois and the surrounding states by U.S. News & World Report. For more information or to make an appointment with a cardiovascular specialist, visit heart.nm.org or call (312) NM-HEART.
Cision View original content with multimedia:http://www.prnewswire.com/news-releases/northwestern-memorial-hospital-surgeon-first-in-illinois-to-repair-damaged-aorta-with-frozen-elephant-trunk-graft-300572065.html
Former National Football League (NFL) players are more likely to have enlarged aortas, a condition that may put them at higher risk of aneurysms, according to a study presented at the annual meeting of the Radiological Society of North America (RSNA), which took place from November 26 to December 1 in Chicago.
The aorta, the largest artery in the body, carries blood from the left ventricle, the heart’s main pumping chamber, to the rest of the body. The short section that rises from the left ventricle and supplies the coronary arteries with blood is called the ascending aorta. Enlargement, or dilation, of the ascending aorta can increase the chances of a life-threatening aneurysm. Risk factors for dilation include high blood pressure, smoking and connective tissue disorders.
Patients whose ascending aortas are more than 4 cm in diameter are generally considered to have dilation, which can progress over time and potentially weaken the wall of the aorta,” says study author Christopher Maroules, MD, formerly of the University of Texas Southwestern Medical Center in Dallas and current chief of cardiothoracic imaging at the Naval Medical Center in Portsmouth, Va.
For the new study, Maroules and colleagues evaluated whether past participation in the NFL is associated with increased prevalence of ascending aortic dilation. The research arose from observations made by the study’s principal investigator Dermot Phelan, MD, PhD, from Cleveland Clinic, who has worked closely over the years with the NFL, studying the cardiovascular health of retired players.
The researchers compared 206 former NFL athletes with 759 men from the Dallas Heart Study who were older than age 40 with a body mass index greater than 20. They obtained imaging data using cardiac gated non-contrast CT, a technique that allowed them to “freeze” the motion of the heart by synchronizing the CT to the electrocardiogram. They also obtained coronary artery calcium scores, a measure of atherosclerotic plaque.
Compared to the control group, former NFL athletes had significantly larger ascending aortic diameters. Almost 30% of the former NFL players had an aorta wider than 4 centimeters, compared with only 8.6% of the non-players. Even after adjusting for age, body mass and cardiac risk factors, former NFL players were still twice as likely as the control group to have an aorta wider than 4 centimeters.
The coronary artery calcium scores were similar in both groups.
“In former NFL athletes, there was a significantly higher proportion of aortic dilation compared to our control group,” says Maroules. “This process is likely not associated with atherosclerosis cardiovascular disease, because when we compared coronary calcium we found no significant difference between the two groups.”
The results suggest that some type of remodeling process occurs in the aorta of athletes who engage in repeated strenuous exercise, according to Maroules.
“It remains to be seen if this remodeling sets athletes up for problems later in life,” he says. “We’re just scratching the surface of this intriguing field, and imaging can play an important role in it.”
Name: Michael McKechnie
Age at time of Dissection: 62
Type of Dissection: Ascending
Date of Aortic Dissection: 21 September 2017
Tell Us Your Story:
From a personal health perspective I cheerfully breezed through the first 62 years of my life. There was the minor tennis injury and the occasional sinus infection but I never had to stay in a hospital and amazingly never missed a day of work. Throughout adulthood daily exercise was imbedded in my lifestyle and I monitored every aspect of my diet. Asking me if I went the gym was like wanting to know if I breathed oxygen, I ate maybe 3 hamburgers a year. I was one of the healthiest people I knew.
This abruptly changed on August 1st 2017.
Two days before my 62nd birthday I had my long past due (oh sorry, Doctor I have just been so busy) wellness check. It was during this routine exam that my primary physician Dr Whelan Culley detected a heart murmur and referred me to a cardiologist named Dr James Neiman. I had no pain or shortness of breath and my history did not suggest alarm but Dr Neiman directed a series of diagnostic tests. After the echocardiogram he asked to see my wife and I at his office as soon as possible and it was then that I heard a group of relatively common words spoken sequentially for the first time. Ascending aortic dissection and aneurysm– what???
Dr Neiman told me to immediately stop exercising and ordered a TEE which not only confirmed the condition but also the precariousness of the aneurysm that somehow contained itself between the two inner layers of my aorta. At this point I was on the fast track and then scheduled to see a heart surgeon, Dr Hartmuth Bittner, (the appointment was delayed twice due to Hurricane Irma, and I filled one afternoon recklessly removing storm panels from my house).
Even though I did some previous research I was still unsure what to expect and was stunned when Dr Bittner outlined what was ahead of me and explained that an extremely intense operation was necessary to replace the damaged section of my aorta. Two days later I spent 12 hours on an operating table. The surgery proceeded normally and was almost over when the right coronary artery unexpectedly burst. Dr Bittner and his team were prepared for just such an emergency, before the operation they took a vein from my leg which they used to bypass the blown artery. This was when my wife and daughters began to feel overwhelmed about the uncanny series of wondrous and inconceivable events that occurred over the last several days.
After 2 weeks in the hospital my homecoming was a treasure, my recovery has had some setbacks but I still expect to eventually be able to resume most of my normal activities. There are no words adequate enough to express the gratitude I have to my incredible doctors and all the professionals at Largo Medical Center.
My survival started with a basic check-up which resulted in an accurate diagnosis before anything really bad happened. Still…why did I not feel any pain? Why did the aneurysm contain itself? (The dissected area was described as being like thin tissue paper). The consensus theory is prior physical condition but I still pause during my recovery and understand that there is a much deeper reason.
As for now I want to hug everyone I see, I’ve fallen in love all over again with my wife and I’m getting reacquainted with God. I feel such a joy in savoring this precious and beautiful gift, it is wonderful to be alive.
Today is one year after my surgery and the first birthday of my new life. Spoiler alert–I feel wonderful!
It was not until the early spring of 2018 when I felt close to normal again, however it became clear that this was an entirely different normal. It evolved into an understanding of a dividing point to a life ‘before 9/21’ and ‘after 9/21′. I came to terms that no matter whatever happens to me the world moves on very quickly and that only I could control how to react to such an incredible change.
The first few weeks of my recovery was a brutal struggle with the physical effects of such an invasive surgery. I could not eat unthickened foods or drink unthickened liquids, I developed thrush, there was numbness in my legs and left arm, just walking to the bathroom was a major accomplishment. And there was the turmoil in dealing with an unexpected emotional aftermath. I felt an intense anxiousness about the unknown. Some mornings I was afraid to just get out of bed, consumed with a fear that I would not get better. I ached for the body I once had. I wept constantly.
As with most recovery success stories it was a support group that helped me through this most difficult period. Encouragement came from my doctors during follow-up visits, the specialists I saw for my cardio rehabilitation, friends who visited me and called regularly. And how rich is a man to be surrounded by a loving family and a devoted wife, my absolute soulmate, who was tirelessly by my side almost every moment throughout this entire ordeal.
I am overwhelmed with gratitude.
Somewhere in mid-life I eased into the comforts of good health and modest prosperity, I rarely thought about my good fortune other than being a just reward for hard work. Over the years I also became spiritually empty and sometimes even began to question the existence of God. Then out of nowhere I’m told I have this life threatening condition, the series of events that lead to my diagnosis and survival were beyond coincidental, I know I was being guided and was called on to accept whatever purpose that is planned for me. So many gifts were right there in front of me but my eyes were closed, I had to be awakened to see and appreciate these treasures.
I will not waste this suffering, I rejoice the beauty and fullness of each day in this wonderful new life.